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2016 Combine Thread

Honestly, I think Doctson is the clear #1 in this class. I do like Treadwell as well though. I think Doctson just gives you a little bit more.
 
Kaiser Toro said:
Based on what I saw of Treadwell in 2014, small hands had little impact on his production, but I prefer Doctson
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Treadwell hands measured 9 1/2" that's not considered small. I agree with you about Doctson, from my point of view Doctson has the best ball skills of all WRs in this draft class, he does things you see Nuk do.
 
Texian said:
Treadwell hands measured 9 1/2" that's not considered small. I agree with you about Doctson, from my point of view Doctson has the best ball skills of all WRs in this draft class, he does things you see Nuk do.
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My mistake on Treadwell's hands, I thought someone stated he had small hands.
 
"Differing opinions" on Myles Jack's progress from season-ending knee injury (via @RapSheet) http://on.nfl.com/1XCPY47

CgP3P3XVAAAG0Dc.jpg
 
Texian said:
David J. Chao, MD ‏@ProFootballDoc 30m30 minutes ago

David J. Chao, MD Retweeted Les Bowen

Unfortunately #JaylonSmith is destined for Day 3 pick with this news. The hope is he can plan 2017 but no guarantee.

David J. Chao, MD added,


Les Bowen @LesBowen
Source says Jaylon Smith's recheck showed drop-foot problem, caused by nerve damage, hasn't improved since the combine.
29 retweets 11 likes
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Don't know if you caught my post of last Friday when Smith's name came up as a high draft candidate.

CloakNNNdagger said:
No one in their right mind would pick Jaylon Smith in the second round. Following his disastrous injury, and with his recent medical re-examination, he will not be playing this year. He has denied any nerve damage all along, but as I posted after the injury occurred, he has sustained a significant injury to his peroneal nerve, which controls elevation (dorsiflexion) of the foot. He actually posted a video with his denial in order to show that he could walk normally without a brace........to "prove" he had no nerve damage. However, after reviewing this VIDEO (scroll down until you find it mid page), I clearly noted that to the untrained eye, the slow walk seemed normal enough.........BUT.......watch his right foot......it points upward (dorsiflexion) as he takes a step forward. Now watch his left foot and you will note the whole foot lifts up, but it does not point upward.........it actually demonstrates the classic peroneal nerve inability to dorsiflex the foot. If this nerve's function does not return, keeping in mind that it supplies movement and sensation to the lower leg, foot and toes , you have a player will not be able to run, stop a cut effectively.
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CloakNNNdagger said:
I just want to clarify my evaluation. We won't know for sure how much of his nerve function could return until ~2 years post injury. But the conclusions drawn from his recent re-evaluation would not be taken as much of a positive.
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Texian said:
Ian Rapoport Verified account ‏@RapSheet Teams drafting in the teens are having internal debates about taking #UCLA LB Myles Jack. Here's why: http://www.nfl.com/news/story/0ap30...ble-to-draft-slide-due-to-concerns-about-knee

David J. Chao, MD ‏@ProFootballDoc 4m4 minutes ago

David J. Chao, MD Retweeted Ian Rapoport

Great job from @RapSheet Fits in with my analysis. My Top 2016 NFL Draft Medical Issues http://shar.es/1e35gm
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The article offers the question of potential need for microfracture surgery. If there is indeed separation of the torn lateral meniscus repair, return to play will almost assuredly further separate and elevate and further tear the lateral meniscus, leaving a larger osteochondral defect. It is hard for me to envision a scenario which does not involve microfracture or the equivalent surgery. In any effect I don't see his immediate 2016 season future in football realistic, and I see his longer term prognosis guarded at this time.
 
CloakNNNdagger said:
The article offers the question of potential need for microfracture surgery. If there is indeed separation of the torn lateral meniscus repair, return to play will almost assuredly further separate and elevate and further tear the lateral meniscus, leaving a larger osteochondral defect. It is hard for me to envision a scenario which does not involve microfracture or the equivalent surgery. In any effect I don't see his immediate 2016 season future in football realistic, and I see his longer term prognosis guarded at this time.
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That's good enough for me.
 
Now what is being reported is that Jack's problem is complicated by a condition that sounds like osteochondritis dessicans. In this condition, it is actually that because the bone has circulation problems and either a segment of bone with its overlying articular cartilage breaks away, or the articular cartilage cannot remain stuck to the bone that essentially deteriorates the bone and replaces it with an inferior bed (for the overlying articular cartilage) of poorly adherent fibrous tissue. This condition can actually begin in childhood and if caught early can be treated with several approaches............the most effective including very extended rest. Some feel that this is of congenital origin, but many feel that that more than half can be attributed to repetitive trauma to the knee from activities, esp., sports beginning as a juvenile. If not caught then and tended to, degenerative disease will become obvious and progress in the collegiate and NFL player. If Jack has significant degenerative disease already, it is not from his meniscus injury. It had to have started before then. But his prognosis has certainly been further complicated by his recent meniscus injury/surgery.
 
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CloakNNNdagger said:
Now what is being reported is that Jack's problem is complicated by a condition that sounds like osteochondritis dessicans. In this condition, it is actually that because the bone has circulation problems and either a segment of bone with its overlying articular cartilage breaks away, or the articular cartilage cannot remain stuck to the bone that essentially deteriorates the bone and replaces it with an inferior bed (for the overlying articular cartilage) of poorly adherent fibrous tissue. This condition can actually begin in childhood and if caught early can be treated with several approaches............the most effective including very extended rest. Some feel that this is of congenital origin, but many feel that that more than half can be attributed to repetitive trauma to the knee from activities, esp., sports beginning as a juvenile. If not caught then and tended to, degenerative disease will become obvious and progress in the collegiate and NFL player. If Jack has significant degenerative disease already, it is not from his meniscus injury. It had to have started before then. But his prognosis has certainly been further complicated by his recent meniscus injury/surgery.
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Ian Rapoport keeps saying he has fluid under the patella and some think he won't see his 2nd contract. Make sense?
 
Texian said:
Ian Rapoport keeps saying he has fluid under the patella and some think he won't see his 2nd contract. Make sense?
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Yes. Fluid is created with articular cartilage damage. The back of the patella basically abuts near the femoral bone condyles that are the typical site of osteochondritis dessicans lesions. Fluid can also be present from damaged meniscus, as well as degenerative patellar-femoral arthritis.

upload_2016-4-29_18-25-33.jpeg (Overlying patella missing)
 
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