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Rest always helps....................but for soft tissue injuries, it takes a LONG rest.........and even then they very likely to become aggravated within the same season (and into others as has been the case with Paul)........not to mention the compensatory injuries that will almost assuredly occur while the tissues are trying to heal.
can you explain why that is the case?
The most common knee ligament injury occurs to the Medial Collateral Ligament, since tackling in football usually occurs from an outside to inside angle (like the football and hockey puck examples below). The medial meniscus, located on the inside of the knee, is more of an elongated "C"- shape, as the tibial surface is larger on that side. The medial meniscus is more commonly injured because it is firmly attached to the medial collateral ligament and joint capsule.
Ahh, got it. thanksThe most common knee ligament injury occurs to the Medial Collateral Ligament, since tackling in football usually occurs from an outside to inside angle (like the football and hockey puck examples below). The medial meniscus, located on the inside of the knee, is more of an elongated "C"- shape, as the tibial surface is larger on that side. The medial meniscus is more commonly injured because it is firmly attached to the medial collateral ligament and joint capsule.
The lateral meniscus, on the outside of the knee, is more circular in shape. The lateral meniscus is more mobile than the medial meniscus as there is no attachment to the lateral collateral ligament (it is directly attached to the proximal fibula) or joint capsule.
The medial meniscus bears up to 50% of the load applied to the medial (inside) compartment of the knee. The lateral meniscus absorbs up to 80% of the load on the lateral (outside) compartment of the knee. When basketball players jump, they usually come down on the outside leg, and usually place an inordinate amount of load on the lateral meniscus. In fact, while running, these forces on the knee increase up to 6 - 8 times body weight, there are even higher forces when landing from a jump. When such skewed concentrated compression force distribution comes down on the lateral meniscus with the knee somewhat flexed (as when a basketball player buffers his landing off of jumps) especially with the slightest of twist, the lateral meniscus is high risk to tear.
"Found no wrong doing"...................there is no way that the medical staff did not have immediate access to the replays of Savage's post-concussion response. The NFL and NFLPA sold their soul in looking the other direction for such an irresponsible conclusion.
I didn't see it in the stadium but they show replays so infrequently I was probably looking at my phone to monitor the game day thread.
or after
I assure you that both Clowney and Watt already have some areas of bone on cartilage where one side is missing some articular cartilage. The bare bone (which many times develops spicules) and irregularities in the damaged articular cartilage and/or the meniscus (which encircles the articular cartilage on the tibial side) on one side will then wear down the articular cartilage on the other side.
Clowney's and Watt's knee injuries/surgeries have left both looking at knee replacements when their careers are over.
This injury is too complicated to cover in more detail than I've done in multiple previous posts. But to answer some of your specific questions:
The plates and screws are always placed through the sides of the bone to create compression in order to bring back the bones into the best alignment possible. Screws and plates are never positioned in the top articular surface of the tibia, so cannot by themselves create articular or meniscus wear. No doubt, with multiple fractures, this realignment will be imperfect at best (not like the flawless precise pic below).............and the overlying articular cartilage is disrupted, and along with the meniscus damage (both structures having very poor healing properties) does not fair well for return to previous performance levels...........and many time for return to play at all. Watt's lay description of his injuries for sure does not present a pretty picture for the future. Short of a microfracture procedure or knee replacement there is little that can be done to "rebuild" the joint surfaces of the knee.
Hope that this gives you a better understanding of what is probably going on with Watt.
This injury is too complicated to cover in more detail than I've done in multiple previous posts. But to answer some of your specific questions:
The plates and screws are always placed through the sides of the bone to create compression in order to bring back the bones into the best alignment possible. Screws and plates are never positioned in the top articular surface of the tibia, so cannot by themselves create articular or meniscus wear. No doubt, with multiple fractures, this realignment will be imperfect at best (not like the flawless precise pic below).............and the overlying articular cartilage is disrupted, and along with the meniscus damage (both structures having very poor healing properties) does not fair well for return to previous performance levels...........and many time for return to play at all. Watt's lay description of his injuries for sure does not present a pretty picture for the future. Short of a microfracture procedure or knee replacement there is little that can be done to "rebuild" the joint surfaces of the knee.
Hope that this gives you a better understanding of what is probably going on with Watt.
There is not much to hear, less than 2 month after the injury. Although he is still using TWO crutches with his boot.
Longhorn NetworkVerified account@LonghornNetwork
What condition are you referring to? Looks like Schaub in one eye.[Thought this post would be more appropriate in this thread]
After injuries, Texans' Watson, Foreman gear up for return
(VIDEO)
A frame pulled from the video:
There is no way Watson's condition is not affecting his visual fields if not corrected medically (if it can be) for games. Since this is a type of condition I have routinely performed surgeries for, I have to wonder why he has not had it addressed. I could swear that I posted about this back in April when I noticed the condition, but I can't seem to find it.
Ptosis surgery is typically performed under local anesthesia, because there needs to be cooperation on the part of the patient (opening and closing the eyes while checking the patient also in a sitting position) to determine the correct amount of correction in attempt to avoid significant over or under correction. There is an element of intrasurgical "trial and error" tightening and loosening until the optimal position is accomplished.
Think you are a bad-azz, don't ya ? That's a snap, but watchin that needle swoop in for a cataract op is much more dicey.
We've got a smilie for this post:
My family has a history of dermatochalasis (excess of skin in the upper eyelid) and my uncles have had multiple surgeries over the years to fix it. Is the eye thing with Watson similar?
Depending on how much it is comes down or doesn't come down over the pupil or how much or how little the muscle can lift the eyelid off of the pupil (especially when trying to gaze upwards), determines its functionality or lack thereof.
Looks like he has that Matt Schaub eye
But Schaub has two of them lol
I certainly don't disagree with the ACL situation. But if you are not interested in a valid observation about a very important aspect of an elite quarterback's tool chest
No. Adrenaline can't have that effect on the eyelid. Its release has an effect on the pupil.......it will simply cause it to dilate.
well dang thanks a whole lot for the mention of the scalpel coming right at MY eye! shudder
You're just pissst that you can't blame McNair or Smith for it.
Define "over time".
In young children, it can occur in terms of over weeks. In an adult, I have seen it occur as soon as 1 year following the diagnosis of severe ptosis. In other cases after several years.
